Course sheet · 48 cases

Case table

Every case of the book with its device printout. Hover over a blurred cell to show the true diagnosis, the steps of the recording or the clinical pearl; on a touch screen, tap it.

Printouts are the LATITUDE™ / ZOOM™ reports the cases were drawn from; the patient’s name and full date of birth are covered. “Book p.” gives the case’s pages in the printed book (paperback, English edition).

One teaching day

9:00–17:00, 2 h of breaks, 7.5–18 minutes per case: the 26 cases in bold, the most important of each chapter.

  1. 09:00–10:30 1 · Counters (5 × 18 min) 1-1 1-2 1-3 1-7 1-8
  2. 10:30–11:00 Coffee break
  3. 11:00–12:30 2 · Therapies (5 × 18 min) 2-6 2-7 2-10 2-11 2-15
  4. 12:30–13:30 Lunch
  5. 13:30–15:30 3 · Discrimination (8 × 15 min) 3-1 3-2 3-4 3-5 3-6 3-7 3-10 3-11
  6. 15:30–16:00 Coffee break
  7. 16:00–17:00 4 · Oversensing (8 × 7.5 min) 4-1 4-3 4-4 4-5 4-6 4-7 4-8 4-9
Download .xlsx.csv48 of 48 cases
#CasePDFFig.Book p.True diagnosisStepsClinical pearl
1-109:00Detection and treatment of a VF episodesame printout as 2-1PDF10–17
VF — appropriate shock
ICM, CRT-D, 1 coil. SR + BiV pacing → fast PVT (2 VT cycles, then VF) → V-Epsd: 8/10 VT zone (2 VT + 6 VF) → 8/10 VF zone: VF duration (5 s) overrides VT → V-Detect → charge ≈10 s (a few undersensed beats, 6/10 kept) → end of charge: 135 ms refractory, 1st beat uncounted, 2/3 fast after the 500 ms window → 41 J synced to R (HVI 76 Ω) → 500 ms post-shock refractory → SR.
An episode starts with 3 consecutive fast cycles, then 8/10 fast (V-Epsd); 6/10 must hold through the duration. Durations: initial, post-detection (VF zone fixed at 1 s), post-shock.
1-209:18Detection and treatment of a VT episodePDF18–23
MMVT — appropriate ATP
ICM, CRT-D. AS + BiV → regular MMVT w/ AVD → V-Epsd after 8/10 VT, VT initial duration 12 s → V-Detect, V>A true → burst of 8 pulses (BiV) → SR.
Probabilistic counters (8/10 to detect, 6/10 during the duration) suit VF, where low, variable signals are partly undersensed, as well as regular VT where every cycle is sensed.
1-309:36Non-sustained ventricular tachycardiaPDF24–29
NSVT — no therapy
DCM, CRT-D, frequent NSVT. AS + BiV → MMVT w/ AVD, VT cycles alternating with PVC-classified beats → 3 consecutive VT cycles open the 8/10 search → V-Epsd (8/10 VT), 12 s duration starts → spontaneous termination → 5 slow cycles: 6/10 lost → V-EpsdEnd 10 s later, no Rx.
Long initial durations (VT zone 12 s, VF zone 5 s) or high zones let NSVT stop on its own; the episode then ends when 8/10 is not met again for 10 s (no therapy delivered).
1-4Change of detection zonePDF30–35
VT accelerating into VF zone — ATP then shock
ICM, CRT-D. AS + BiV → MMVT w/ AVD, VT-zone then VF-zone cycles → V-Epsd VT (5 VT + 3 VF), VT initial duration 2.5 s → 8/10 VF, VF duration starts → VT duration ends but VF 6/10 still valid: VT Rx held → 5 VT cycles: VF 6/10 lost → burst (1st VT Rx) fails → redetection 8/10 VT and VF, post-redetection 1 s each → VT V-Dur, VF still running → VF duration ends → charge → divert window → 41 J on 2nd fast cycle → SR.
Each zone has its own 8/10 window and duration, running in parallel; after ATP for VT, acceleration into the VF zone gets the 1 s VF redetection, not the long initial duration.
1-5Spontaneous termination and shock diversionpatient 2 only (no 5a)PDF36–42
Self-terminating polymorphic VT — charge diverted
Pt 1, VR ICD: PVT → V-Epsd VT-1 (8/10) → 8/10 VT then VF, durations → end of VF duration → ATP QC fails → charge → spontaneous termination → 6/10 lost → charge diverted. Pt 2, DR ICD: same; PVC in the V noise window after AP ([VS]) → ATP QC fails → charge → termination → end of charge, 500 ms window, 1st beat uncounted (135 ms) → 2 of 3 slow → charge diverted.
"Shock not reconfirmed" Off: charging stops on 5 slow beats out of 10 (6/10 lost). On: once charging starts, the shock is always delivered (first R wave after 500 ms).
1-6Diversion window at end of a chargePDF43–47
VF (+ AF) — appropriate shock
ICM, CRT-D, pAF. AF + BiV → very fast PVT in VF zone → V-Epsd (8/10 VF), initial duration → V-Detect → charge 11 s (max output) → end of charge: 135 ms refractory, 1st beat uncounted → next 2 VF beats fall inside the 500 ms divert window → shock synced to the 3rd fast beat (41 J, 64 Ω) → VF + AF end → 500 ms post-shock, 1st A and V beats uncounted.
After charge end comes a 500 ms diversion window (its first 135 ms refractory); a reconfirmed shock needs 2 of the next 3 intervals faster than the lowest zone; 500 ms refractory after any shock.
1-709:54Undersensing and charge diversionPDF48–53
Induced VF with undersensing — diverted, then shock
Brugada, VR ICD, septal lead (R 3.5 mV), induction at 1 mV: low-energy T-wave shock after 8 pulses at 200 bpm → low-voltage PVT/VF → V-Epsd (8/10 VF) → 1st V event undersensed → 1 s duration, V-Detect, charge → end of charge: undersensing, 2/3 fast not met → Dvrt (false “termination”) → new 8/10, post-detection duration → V-Detect → very short charge (capacitors still charged) → not reconfirmed (2nd charge of an episode is never diverted) → 21 J → SR.
AGC: after refractory + 15 ms the threshold drops to 75% of the last peak (mean peak if paced), then 7/8 per step down to Min or the programmed value; only one charge per episode can be diverted.
1-810:12End-of-episode criteria following an electric shockPDF54–67
VF — 3 shocks; end-of-episode criteria
ICM, CRT-D. AP + BiV → VF → VF duration 5 s → charge → 41 J on 2nd fast beat → VF restarts at once → redetection 8/10 VT (1 s) and VF (post-shock 1 s, fixed) → V-Detect → charge → 2nd shock after 500 ms, the 30 s end-of-episode clock starts → recurrence <30 s = same episode → shock → SR … later VF: V-EpsdEnd (30 s without 8/10, counters reset) → new episode: 8/10 VT + VF → ATP QC (1st Rx of a new episode) fails → shock → recurrence → 1 s post-shock duration → shock → SR.
V-Epsd End when 8/10 is not met again for a fixed time: 10 s after no therapy or ATP only, 30 s after any shock. 8/10 within it = same episode: 1 s post-shock duration, next shock.
2-1VF episode correctly detected and treated with maximum output shocksame printout as 1-1PDF70–77
VF — appropriate shock
ICM, CRT-D. SR + BiV → fast PVT (1 VT, then VF) → V-Epsd 8/10 VT (1 VT + 7 VF) → 8/10 VF, VF duration 5 s takes precedence → V-Detect → charge ≈9 s (a few RVS undersensed, too sparse to divert) → 135 ms, 1st beat uncounted, 2/3 fast after 500 ms → 41 J on 2nd beat (54 Ω) → no beat: V pacing 2 s after the shock → SR.
Up to 8 shocks in the VF zone; shocks 3 to 8 are always 41 J. Maximum energy from the first shock is reasonable (IIa) unless a lower energy proved effective at induction.
2-2VF episode with low-amplitude shock in a childPDF78–82
VF in a child — low-energy shock
13-y/o post-SCA, VR ICD, recurrent VF. SR (VS) → very fast PVT, every beat in VF zone → V-Epsd (8/10 VF), initial duration programmed at 1 s → charge 2.7 s → 135 ms + uncounted beat, then 2 fast (VF, VT) after 500 ms → 14 J synced (33 Ω) → SR.
Children: first shock about 1 J/kg (2-5 J in neonates), usually one high VF zone, close remote monitoring; inappropriate therapies are commoner (lead fracture, sinus tachycardia, TWOS).
2-3Multiple electric shocks in the VF zonePDF83–94
VF — 5 max-output shocks
ICM, CRT-D. Slow VT → accelerates into VF zone → end of duration, charge → degenerates into VF → 41 J on 2nd fast beat → organized VT restarts → redetection 8/10, VF 1 s → charge → shock after 500 ms (no 2/3 check after a shock) → VT/VF again → same sequence → shocks 3, 4, 5 (41 J each) → termination.
A high DFT seen in spontaneous VF is an emergency. Programmable: energy, vector and polarity (biphasic, tilt fixed). With a single-coil lead only RV coil → can delivers a shock.
2-4VT in the VF zone and high defibrillation thresholdpaper book (EN): item 4 has no badge on the trace; from badge 4 on, badge n = item n+1PDF95–105
MMVT degraded to VF by shock — high DFT
ICM, DR ICD. Organized VT in VF zone → 8/10 VT then 8/10 VF; VT duration ends, VF still running → VF duration ends → ATP QC fails → charge → 41 J → 2 slow beats then very fast disorganized VF → 1 s redetection → 2nd shock → same pattern (shock, 2 slow beats, VF) → 3rd, 4th shock → slow VT persists. High DFT: CS coil added.
The DFT is not a fixed value: a shock can turn stable VT into VF and repeated maximum shocks may fail; an extra coil (e.g. coronary sinus) widens the shock field.
2-5VF, device shock with AF inductionPDF106–112
Fast VT — appropriate shock, shock-induced AF
ICM, DR ICD. Fast VT w/ AVD in VF zone → V undersensing but 6/10 kept → ATP QC fails → charge → 41 J → VT ends → A beat sensed in refractory → A pacing in the atrial vulnerable period → AF induced.
Ventricular shocks are synchronized to the R wave whatever the atrial phase and can induce AF, more often with low energy and dual-coil shocks; induced AF is usually short.
2-611:00ATP burst in the VF zonePDF113–126
Recurrent MMVT in VF zone — ATP QC then shocks
DCM, DR ICD. SR → MMVT in VF zone → 8/10 VF, 1 s duration → V-Detect, rate (last 4 cycles) 229 <250 → ATP QC (8-pulse burst) → 1st beats uncounted, 2 of 3 slow → charge diverted → V-EpsdEnd → same VT recurs → ATP QC → 2/3 fast → charge → 41 J on 2nd fast beat → SR → VT recurs <30 s (same episode) → no 2nd ATP QC (1 per episode) → charge → 41 J → SR.
ATP first (a burst rather than a ramp, class I) up to about 230 bpm cuts shocks; after the burst, 2 of 3 fast intervals start the charge, 2 of 3 slow or a 2 s pause divert therapy.
2-711:18ATP burst in the VF zone with accelerationPDF127–132
MMVT accelerated by ATP QC — shock
Severe CM, perm. AF, CRT-D. MMVT in VF zone → 8/10 VF, 1 s duration → V-Detect, rate 226 <250 → ATP QC (8 pulses, 88 % coupling) → 1st beat in refractory ([VS]), 2nd uncounted → 2/3 fast → charge → VT accelerates (fast, low voltage) → end of charge, 500 ms window → 31 J on 2nd fast beat → SR.
ATP Quick Convert (VF zone, before charging, up to 250 or 300 bpm) is a fixed burst of 8 at 88%; if it fails or accelerates the arrhythmia, the shock comes later and succeeds less often.
2-8Undersensing, rate calculation error and ATP burst in the VF zonePDF133–138
PVT/VF with undersensing — futile ATP QC, shock
Idiopathic VF, DR ICD. AP (sensor rate) + VS → very fast PVT → undersensed beat classified VS → V-Detect: rate over the last 4 cycles 225 <250 (falsely low) → ATP QC, no V capture → charge → shock on the 3rd VF beat (the 2nd falls inside the 500 ms window) → 41 J → SR.
The rate that allows ATP is the mean of the 4 cycles before V-Detect, so undersensing can let ATP fire in fast VF; evidence for ATP above 250 bpm is weak.
2-9Oversensing and anti-tachycardia pacing in the VF zonePDF139–144
Lead noise (oversensing) — inappropriate ATP
ICM, DR ICD (box change, old RV lead). AP-Sr/VP-Sr → noise of variable amplitude on V sense, shock EGM clean → VF counter fills (V-Epsd) → intermittent: at end of duration rhythm = sinus, no Rx → noise again → VF counter → ATP QC (2 of 8 pulses capture) → noise persists, 2/3 fast → charge → noise stops: 6/10 lost → charge diverted → noise resumes → VT counter (V-Detect) → ATP, no capture.
Lead noise can trigger very fast inappropriate ATP; systematic ATP up to 300 bpm is unproven and can be pro-arrhythmic when inappropriate.
2-1011:36Anti-tachycardia pacing in the VT zonePDF145–149
MMVT — ATP success
CRT-D. AS + BiV → MMVT w/ AVD in VT zone → V-Epsd (8/10 VT/VT-1) → initial duration 12 s → V-Detect, V>A → burst of 10 pulses (BiV) → SR.
ATP terminates about 90% of VTs below 200 bpm and works up to about 240 bpm; sequences can be programmed empirically, sparing shocks, symptoms and battery.
2-1111:54Anti-tachycardia pacing in the VT zone and post-detection durationPDF150–160
VT — multiple ATP sequences
CRT-D. AS + BiV → MMVT w/ AVD → V-Epsd, 12 s → V-Detect (V>A) → burst of 10 → SR → same VT before V-EpsdEnd → 8/10, post-detection duration 1 s → 2nd burst (same episode) → works, quick recurrence → bursts 3, 4 → ramps 1–3 (−10 ms per pulse, min 200 ms) → NSVT, 8/10 not met.
VT recurring before V-Epsd End gets the VT-zone redetection duration (here 1 s) and is paced after a few cycles: lengthen it (programmable in the VT zone only) to let NSVT stop.
2-12Progression of therapies in the VT zonePDF161–173
VT — ATP then 11 J shock
CRT-D. AS + BiV → MMVT w/ AVD → V-Epsd, 12 s → V-Detect (V>A) → burst 1 fails → 8/10, 1 s → burst 2 fails → bursts 3, 4 → ramps 1, 2 → ramp 3 works → VT recurs before V-EpsdEnd (same episode) → ramp 4 → next Rx = shock: charge → 11 J on 2nd fast beat after the divert window (2/3 fast) → SR.
Recurrence before V-Epsd End moves on to the next therapy in sequence. Burst settings: 5-15 pulses, coupling around 88%, a minimum interval limit, 5 V / 1 ms output.
2-13Low-amplitude shock and acceleration of arrhythmiaPDF174–185
VT — low-energy shock accelerates, 41 J shock
CRT-D. MMVT w/ AVD in VT zone → burst 1 (8 pulses) fails → burst 2 (+1 pulse) → bursts 3, 4 → ramps 1–3 → next Rx = shock: charge → 11 J on 2nd fast beat (2/3) → VT accelerates into VF zone → VF detection, charge → 41 J → SR.
A first VT-zone shock of 10-15 J charges quickly and often works, but can accelerate the VT (upper limit of vulnerability).
2-14Maximum output shock in the VT zonePDF186–194
VT — ATP fails, max shock
VR ICD. Probable VT (morphology change on the shock EGM) → end of duration: burst of 10 fails → bursts 2, 3 → ramps 1–3 → next Rx = shock: charge → 41 J on 2nd fast beat after the divert window (2/3 fast) → SR.
A first VT-zone shock at maximum energy lowers the pro-arrhythmic risk and raises first-shock success, at the cost of a longer charge.
2-1512:12Anti-tachycardia pacing in the VT zone with acceleration into the VF zonePDF195–210
VT — ATP accelerates into VF zone, shock
CRT-D. VT w/ AVD in VT zone → end of duration: burst of 10 → VT accelerates into VF zone → VF detection, charge → becomes polymorphic → 41 J → SR.
ATP accelerates VT or degenerates it into VF in about 1-5% of cases: shocks must always be programmed as back-up.
3-113:30Single-chamber discrimination, Rhythm ID and a ventricular tachycardia episodePDF211–216
VT — appropriate ATP (RID−)
ICM, VR ICD, RID thr 94 %. Abrupt, stable tachycardia >200 bpm, shock-EGM morphology ≠ SR → every beat uncorrelated (U, 0 %) → V-Epsd (8/10 VT), 12 s → V-Detect: RID− (0/10 correlated, <3/10) → VT → burst → SR.
Boston discriminators act only in VT/VT-1 zones (VF zone: rate alone). Program them only if conducted SVT is possible; judge them by sensitivity for VT (near 100%) and specificity.
3-213:45Single-chamber discrimination, Rhythm ID and an atrial fibrillation episodePDF217–221
AF — therapy correctly withheld (RID+)
HCM, VR ICD, RID 94 %. Irregular tachycardia, narrow shock EGM; VT-1/VS alternate, every beat correlated (C) → V-EpsdEnd of a previous episode (8/10 not met) → V-Epsd (8/10 VT-1) → V-Dur: RID+ (≥3/10 correlated) → SVT, Rx withheld; beat-by-beat analysis goes on.
Rhythm ID compares 8 points of the shock-channel QRS with a sinus template: 3 or more of 10 cycles correlated = SVT (RID+, therapy withheld), fewer = VT (RID-); VF-zone cycles never correlate.
3-3Single-chamber discrimination, Rhythm ID and sinus tachycardiaPDF222–226
Sinus tachycardia — therapy correctly withheld (RID+)
ICM, VR ICD, RID 94 %. Regular tachycardia, narrow shock EGM, gradual acceleration VS → VT-1, every beat correlated → V-EpsdEnd (previous episode) → V-Epsd (8/10 VT-1) → V-Dur: RID+ → Rx withheld → rate slows (VS), still correlated.
The template is refreshed passively (every 2 h) or actively (every 28 h, pacing modified); at follow-up, check that the match stays well above the threshold (nominal 94%).
3-414:00Single-chamber discrimination, Rhythm ID and correlation percentagePDF227–237
Sinus tachycardia — inappropriate therapies (RID threshold too high)
ICM, VR ICD, RID 94 %. Regular narrow tachycardia, gradual acceleration VS → VT-1, correlated → VT-1 duration ends (no Rx in VT-1) → faster, VT zone: RID+ → withheld → V-Detect: only 2/10 correlated → RID− → “VT” → inappropriate burst → redetection: no morphology analysis after a Rx → every VT-zone Rx delivered. Fix: lower the 94 % threshold (83 %: sens 100 %, spec 79 %).
Nominal RhythmMatch 94%: 100% sensitivity but about 41% specificity (morphology checked at initial detection only); 83% kept 100% sensitivity and raised specificity to 79%.
3-514:15Single-chamber discrimination, Rhythm ID and SRDPDF238–248
AF — inappropriate shocks (SRD expired)
ICM, VR ICD, RID 94 %. Irregular narrow tachycardia (AF), VT-1/VS alternate, correlated → V-EpsdEnd (previous) → V-Epsd (8/10 VT-1/VT) → V-Dur: RID+ → withheld → SRD timer expires (SRD marker) → VT-zone Rx despite RID+ → successive Rx up to 5 shocks. Fix: SRD Off or several minutes.
SRD delivers therapy after a set time even when discriminators say SVT; guidelines advise turning it off or setting it to several minutes.
3-614:30Dual chamber discrimination and V>APDF249–253
VT with AV dissociation — appropriate ATP (V>A)
ICM, CRT-D. AS + BiV → abrupt regular tachycardia w/ AVD (V 156 > A 62 bpm) → V-Epsd: beats uncorrelated (U, <94 %) → V>A true (V ≥10 bpm faster over the last 10 intervals) → VT (V>A overrides RID) → V-Detect → burst → SR.
V>A (mean of the last 10 V vs 10 A intervals, at least 10 bpm faster) overrides every other discriminator: True = VT, therapy.
3-714:45Dual-chamber discrimination, atrial refractory periods and V>A criterionPDF254–263
Sinus tachycardia — inappropriate ATP (false V>A)
ICM, DR ICD, RID. Probable 1:1 ST, VS/VT-1 alternate → 8/10 met → end of duration: stable + correlated → no Rx → beat by beat: R-wave timing varies → “unstable”, still correlated → stable again → PAC → 2nd PAC falls in the A refractory period after V sense (long PR) → not counted → false atrial slowing → V>A true (overrides RID) → bursts 1–5, inappropriate → sinus rate falls below VT-1 → end.
Undersensed atrial events (an atrial extrasystole in the refractory period) can make V>A falsely True and override correct morphology; V>A needs good atrial sensing, otherwise turn it off.
3-8Dual-chamber discrimination and tachycardiaPDF264–269
Sinus tachycardia — therapy correctly withheld
DCM, DR ICD, RID + atrial discrimination On. Probable conducted ST, VS → acceleration into VT-1 → correlated (>94 %) → V-Epsd (8/10 VT-1) → end of duration: V>A false → RID+ (all correlated) → withheld → slows to VS. Logic: step 1 V>A, step 2 RID, step 3 AFib/stability.
Dual-chamber Rhythm ID works in steps: V>A first (True = VT), then morphology (3 or more of 10 correlated = SVT), then stability and the AFib rate threshold.
3-9Dual-chamber discrimination and atrial tachycardiaPDF270–274
Atrial tachycardia — therapy correctly withheld
ICM, DR ICD, RID. Probable AT w/ variable AV conduction, VS/VT alternate, correlated → V-Epsd (8/10 VT) → end of duration: V>A false, RID+ → SVT, withheld → rate slows (VS), 6/10 VT lost.
1/1 atrial tachycardia or flutter fools onset and stability (abrupt and stable, like VT); morphology-based Rhythm ID is preferred in these patients.
3-1015:00Dual-chamber discrimination and dual tachycardiaPDF275–279
Dual tachycardia (AF + VT) — appropriate ATP
Mitral CM, DR ICD. AF w/ fast irregular conduction → sudden regular V rhythm, new EGM morphology (VT) → 3 consecutive VT cycles: atrial intervals classed against the AFib threshold → V-Epsd (8 consecutive VT) → 10 cycles to V-Detect: correlation → V>A false, RID− → AF+ (6/10 then 4/10 fast A) + stable → dual tachycardia → treat → burst of 10 fails → V-Detect on rate only (no discrimination after a Rx) → 2nd burst → VT ends, AF goes on.
With V>A false and morphology uncorrelated, stability and the AFib rate threshold decide: stable, or no fast atria → therapy; unstable with fast atria → inhibition.
3-1115:15Dual-chamber discrimination and AF with aberrant conductionPDF280–282
AF/AT with aberrancy — therapy correctly withheld
ICM, DR ICD. Fast atrial activity (probable AT/AF) w/ fast variable conduction, VS/VT/VF cycles → V-Epsd (8/10) → end of duration: V>A false, RID− (aberrancy), AF+ (A rate > AFib threshold), unstable (>30 ms) → SVT (aberrantly conducted AF) → withheld → beat by beat, same diagnosis.
Dual tachycardias and AF with aberrancy are the hardest: V>A cannot decide; with uncorrelated vectors and fast atria, stability makes the call (stable = VT).
3-12Dual-chamber discrimination and discrimination error due to signal variability on the shock channelPDF283–290
SVT — inappropriate ATP + 6 shocks
HCM, DR ICD. Regular 1:1 tachycardia (AS-VS) → A (AF) + V (VT) acceleration → V-Epsd (8 VT); shock-EGM signal variable (myopotentials) → end of duration: V>A false, stable, AF+, RID+ → withheld → still RID+ → shock-EGM noise: RID− + AF+ + stable → “dual tachycardia” → 1st Rx → redetection (no discrimination) → every VT-zone Rx (ATP + 6 shocks).
Morphology is read on the shock channel: myopotential noise there decorrelated SVT cycles and caused inappropriate therapy.
3-13Dual-chamber discrimination and discrimination error due to inappropriate programming of the correlation percentagetwo episodesPDF a PDF b291–297
1:1 atrial tachycardia — inappropriate ATP (RID threshold too high)
ICM, CRT-D, RID 94 %. Ep 1: regular 1:1 tachycardia, 1st fast beat atrial (AT) → V-Epsd (8 VT/VF), correlated → end of duration: stable, AF+, RID+ → withheld → RID− (<3/10 correlated) → “dual tachy” → burst → ends. Ep 2: same 1:1 AT → end of duration: RID−, stable → bursts/ramps → V stops spontaneously. Fix: threshold 88 %.
A correlation threshold set too high misclassified atrial tachycardia; 88% would have prevented the inappropriate therapies.
3-14Dual-chamber discrimination, atrial tachycardiaPDF298–300
1:1 atrial tachycardia — therapy withheld (gradual onset)
ICM, wide QRS, CRT-D, Onset/Stab. Fast 1:1 rate at the VT-zone edge (RVS) → slight acceleration into VT zone (<375 ms) → 8/10; onset gradual (Gradl) → end of duration: stable + gradual → Rx withheld (read as ST) → beat by beat: still stable, withheld. True dx 1:1 AT; fix: raise the VT zone or use RID.
Onset (lowest zone only) withholds therapy for a gradual rate rise; 1/1 AT hovering at the zone limit defeats it: raise the VT zone above the clinical AT or rely on morphology.
3-15Dual-chamber discrimination and atrial fibrillationPDF301–308
Conducted AF — inappropriate ATP + shock (stability)
ICM, CRT-D, Onset/Stab, stability thr 20 ms. Irregular conducted AF → V-Epsd (Gradl) → end of duration: AF+ and unstable → withheld → conducted AF stabilises → “stable” → bursts → ramps → max shock, all inappropriate → the shock ends AF. Fix: stability 30–40 ms.
Conducted AF can stabilize and pass the 20 ms nominal stability criterion; tune stability and the AFib rate threshold (170 bpm nominal; too low risks therapy for sinus tachycardia).
3-16Dual chamber discrimination, atrial tachycardiatwo episodesPDF a PDF b309–315
1:1 atrial tachycardia — ATP (Onset/Stab) vs withheld (RID)
ICM, CRT-D. Ep 1 (Onset/Stab): regular 1:1 tachycardia, A beats classed AF → V-Epsd (Gradl) → end of duration: AF+ and stable → burst (inappropriate) → SR. Ep 2 (RID): identical 1:1 AT → V-Epsd → end of duration: RID+ → withheld → spontaneous termination (favours AT).
Onset/Stability handles 1/1 AT poorly (fast atria and a stable rhythm mean therapy); the same tachycardia may be inhibited another time: programming is a trade-off.

Discrimination rules (Boston Scientific)

Discrimination works only in the VT zones (VT, VT-1), at the end of the initial duration; in the VF zone rate alone decides. After a first therapy, redetection is on rate only (no discrimination). SRD (sustained rate duration) treats despite an SVT verdict once its timer expires: usually Off (3-5).

Single chamber: Rhythm ID only

  • Each tachycardia beat is compared with the stored sinus-rhythm template: aligned on the sensing EGM, 8 points compared on the shock EGM → % match (RhythmMatch).
  • Match ≥ threshold (nominal 94 %, programmable 70–96 %) = correlated.
  • ≥ 3 of the last 10 beats correlated → SVT, therapy withheld (RID+); < 3/10 → VT, treat (RID−) (3-1, 3-2, 3-3).
  • A beat in the VF zone always counts as uncorrelated.
  • Pitfall: 94 % too high → narrow ST read as VT (3-4); 83 % kept 100 % sensitivity, 79 % specificity.

Dual chamber: Rhythm ID (in steps)

  • 1. V>A: mean V rate of the last 10 intervals ≥ 10 bpm above the A rate → VT, treat. Overrides everything (3-6).
  • 2. RID: ≥ 3/10 beats correlated → SVT, withhold (3-8, 3-9).
  • 3. RID−: AFib rate threshold + stability. AF+ (≥ 6/10 A intervals faster than the AFib threshold, nominal 170 bpm) and unstable (mean RR difference > stability, nominal 20 ms) → conducted AF, withhold (3-11); AF+ and stable → dual tachycardia, treat (3-10); no AF → treat.
  • Pitfalls: a PAC in the atrial refractory period is not counted → false V>A (3-7); noise on the shock EGM → RID− (3-12); threshold too high → 1:1 AT treated (3-13).

Dual chamber: Onset/Stability

  • V>A true → VT, treat (same first step).
  • Onset (lowest zone only): pivot point with the largest drop in cycle length ≥ Onset value → sudden; gradual onset → withhold (3-14).
  • Unstable + AF+ → withhold; sudden + stable → treat; gradual + AF+ + stable → treat (3-15, 3-16).
  • Pitfalls: 1:1 AT looks like VT for onset and stability (3-14, 3-16: Rhythm ID is preferred); conducted AF that stabilises below 20 ms → treated (3-15: 30–40 ms); AFib threshold too low in the young → ST treated.

Guidelines: program discrimination up to 230 bpm; turn it off in complete, permanent AV block.

4-116:00Oversensing of electrocauteryPDF318–321
Electrocautery EMI — no therapy (Monitor only)
ICM, CRT-D, box change, Tachy Mode Monitor only. AS + BiV → electrocautery: fast, chaotic signals of variable amplitude on A, V and shock channels at once (AF, VF markers) → VF-zone episode, no Rx → stops → recurs. Rules: Tachy Off during surgery; asynchronous pacing if dependent; bipolar cautery, short bursts.
Electrocautery: fast, chaotic signals on all 3 channels. Set Tachy Mode off during surgery (asynchronous pacing if dependent); bipolar cautery, short bursts, plate far from the device.
4-2Surgical intervention and VF at the time of reperfusiona = patient 1, b = patient 2PDF a PDF b322–328
EMI + cardioplegia; true VF at reperfusion — shock
Pt 1 (AVR, CPB, Monitor only): no A/V activity (cardioplegia asystole despite pacing), baseline noise → cautery: fast chaotic signals on 3 channels (AF/VF) → VF zone, no Rx. Pt 2 (CABG, Monitor + Therapy): cautery on V + shock channels → stops → true very fast low-voltage VF at reperfusion (imperfect sensing) → detection, charge → 41 J → SR.
Cardiac surgery: cardioplegia shows as asystole despite pacing, VF is frequent at reperfusion. Usual practice: Tachy Mode off, temporary pacing and external defibrillation.
4-316:08Inappropriate therapies during a TAVI procedurePDF329–333
Oversensed pacing artefacts during TAVI — inappropriate ATP
CRT-D, TAVI. AS + BiV → temporary RV rapid pacing: sharp spikes on A, V and shock channels (artefacts, capture seen on the shock EGM) → VT/VF cycles alternate: 8/10 VT met, VF not → end of VT duration → ATP sequences → next Rx = shock: charge → pacing stops, oversensing ends → 2/3 fast not met → charge diverted. Rule: deprogram Tachy for TAVI.
Rapid ventricular pacing during TAVI is oversensed: suspend ICD therapies; LATITUDE can flag 4 or more cycles of 160 ms or less as a non-physiological RV signal.
4-416:15Lead fracture and multiple capacitor chargesa = follow-up reports, b = episodesPDF a PDF b334–337
RV lead fracture — no therapy
VR ICD, LATITUDE Z alert. Fast signals on the bipolar sense EGM, shock EGM clean → variable-amplitude noise, some saturating → episode in VT zone, no Rx → noise ends. Follow-up: pace Z jump >2000 Ω, shock Z stable, many NSVT; interval plot = cloud of very short cycles (near blanking) + slower ones → RV lead fracture.
Lead fracture: intermittent very short, variable cycles plus abnormal pacing impedance (above 2000 Ω) with stable shock impedance; remote monitoring catches it before shocks.
4-516:23Oversensing of the P wave by the defibrillation leadSVG strip, converted to PDFPDF338–340
P-wave oversensing — no therapy
CRT-D. ST → intermittent oversensing of atrial activity by the RV lead (P wave seen on the A channel) → beats classed VS/VT-1/VT/VF → 8/10 VT-1 → NSVT stored, no Rx. Causes: RV lead near the AV junction, coil in RA, integrated bipolar.
P-wave oversensing by the RV lead: displacement to the AV junction (R wave falls) or an integrated bipolar lead straddling the tricuspid valve; lower sensitivity (then check VF sensing) or revise the lead.
4-616:30Oversensing of atrial activity by the defibrillation lead during an episode of atrial arrhythmiaPDF341–342
AF oversensed by RV lead — pause (pacing-dependent)
DCM + CHB, DR ICD, integrated bipolar RV lead (coil = anode). AF correctly detected, mode switch, VP-Sr at sensor rate (dependent) → fast A activity oversensed on V sense (shock EGM: no V activity) → V pacing inhibited, near-pause (presyncope) → oversensing ends. Risk: inappropriate Rx + asystole.
In pacing-dependent patients, oversensed atrial arrhythmia inhibits ventricular pacing (asystole) as well as risking inappropriate therapy.
4-716:38Early displacement of a right ventricular lead into the atriumPDF343–348
RV lead dislodged into RA, AF oversensed — inappropriate shock
CRT-D, day 1. A and RV sensing almost simultaneous, LV later (true V) → AF onset: very fast signals on A, sensed on RV too (intermittently) → sustained RV oversensing → 8/10 VF → end of duration, charge → 6/10 lost → charge diverted → 2nd charge (cannot be diverted) → after 500 ms: inappropriate 41 J → AF and oversensing stop. CXR: RV lead floating in RA; Rx Off until revision.
Early RV lead dislodgement into the atrium: AF was sensed as VF and shocked; suspend therapies until the lead is repositioned.
4-816:45T wave oversensingPDF349–352
T-wave oversensing — inappropriate ATP, charges aborted/diverted
VR ICD, small R wave. SR with intermittent TWOS (R/T alternation) → prolonged TWOS → V-Epsd (8/10) → 1 s duration, charge → PVC + undersensed QRS → undersensing: charge interrupted → 8/10 again → end of duration: ATP QC (inappropriate) → TWOS gone: charge diverted.
T-wave oversensing (rare with Boston) alternates high-frequency R and low-frequency T signals; small R waves favour it, as the AGC then reaches high sensitivity quickly.
4-916:53Oversensing of diaphragmatic myopotentialsPDF353–355
Diaphragmatic myopotentials — no therapy
VR ICD, integrated bipolar at RV apex. Low-amplitude non-physiological signals (crescendo–decrescendo) on V sense only, shock EGM clean → 3 consecutive beats classed VF → EGM stored as an NSVT episode, no Rx. Clues: end of diastole, high auto-sensitivity, apical integrated bipolar lead.
Diaphragmatic myopotentials: low-amplitude, high-frequency noise on the sensing channel only, late in diastole at maximum sensitivity; commoner with integrated bipolar apical leads.

No case matches.

Abbreviations
-Sr
paced at the sensor-indicated rate
8/10, 6/10, 2/3
fast intervals out of the last 10 (to detect / to stay detected), fast beats out of 3 after a charge
AF
atrial fibrillation
AS / AP, VS / VP
atrial / ventricular sensed / paced event
AT
atrial tachycardia
ATP QC
ATP Quick Convert (ATP in the VF zone)
AVC / AVD
AV conduction / AV dissociation
AVR / CABG
aortic valve replacement / coronary bypass
BiV
biventricular pacing
CHB
complete heart block
CPB
cardiopulmonary bypass
CS
coronary sinus
CT
charge time
CXR
chest X-ray
DCM / HCM / ICM
dilated / hypertrophic / ischaemic cardiomyopathy
DFT
defibrillation threshold
Dvrt
charge diverted
EMI
electromagnetic interference
Gradl
gradual onset
HVI
shock (high-voltage) impedance
MMVT / PVT
monomorphic / polymorphic VT
NSVT
non-sustained VT
Onset/Stab
Onset/Stability discrimination
PAC
premature atrial complex
PVC
premature ventricular complex
RID+ / RID−
Rhythm ID correlated (SVT) / not correlated (VT)
Rx
therapy
SCA
sudden cardiac arrest
SR / ST
sinus rhythm / sinus tachycardia
SRD
sustained rate duration
TAVI
transcatheter aortic valve implantation
thr
threshold
TWOS
T-wave oversensing
V-Epsd / V-Detect / V-Dur / V-EpsdEnd
episode start (8/10) / detection at end of duration / end of duration / end of episode
VR / DR ICD
single- / dual-chamber ICD
Z
impedance
Open full size

From Boston Scientific ICD — clinical cases by P. Bordachar, M. Strik, A. Thiyagarajah, S. Ploux. Published by Cardiocases.